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sult of brainstem compression, at which point reversal of the syndrome, even by surgical therapy, is seldom successful Lobar Hemorrhage Bleeding in areas other than those listed above, speci cally in the subcortical white matter of one of the lobes of hemisphere, is not associated strictly with hypertension; it is presented here for ease of exposition Any number of other causes are usually responsible, the main ones being anticoagulation or thrombolytic therapy, arteriovenous malformation (discussed further on), trauma, and, in the elderly, amyloidosis of the cerebral vessels In a series of 26 cases of lobar hemorrhage, we found 11 to lie within the occipital lobe (with pain around the ipsilateral eye and a dense homonymous hemianopia); 7 in the temporal lobe (with pain in or anterior to the ear, partial hemianopia, and uent aphasia); 4 in the frontal lobe (with frontal headache and contralateral hemiplegia, mainly of the arm); and 3 in the parietal lobe (with anterior temporal headache and hemisensory de cit contralaterally) (Ropper and Davis) The smaller hematomas simulate an embolic stroke in the same territory The occurrence of a progressively worsening headache, vomiting, and drowsiness in conjunction with any one of these syndromes is virtually diagnostic, and, of course, a lobar hemorrhage is readily corroborated by an unenhanced CT scan Of our 26 patients, 14 had had normal blood pressure, and in several of the fatal cases there was amyloidosis of the affected vessels (see further on) Also, 2 patients were receiving anticoagulants, 2 had an arteriovenous malformation, and 1 had a metastatic tumor In a series of 22 patients with lobar clots reported by Kase and colleagues, 55 percent were normotensive; metastatic tumors, arteriovenous malformations, and blood dyscrasias were found in 14, 9, and 5 percent of the patients, respectively The role of amyloid angiopathy in lobar hemorrhage in the elderly patient is discussed further on In the localization of an intracerebral hemorrhage, ocular signs are particularly important In putaminal hemorrhage, the eyes are deviated to the side opposite the paralysis; in thalamic hemorrhage, the commonest ocular abnormality is downward deviation of the eyes and the pupils may be unreactive; in pontine hemorrhage, the eyeballs are xed and the pupils are tiny but reactive; and in large cerebellar hemorrhage, the eyes are deviated laterally to the side opposite the lesion and ocular bobbing may occur Although the proper interpretation of this array of clinical data allows the correct diagnosis to be established in most cases, the ancillary examinations described below are de nitive, especially in the diagnosis of small hemorrhages Laboratory Findings Among laboratory methods for the diagnosis of intracerebral hemorrhage, the CT scan occupies the foremost position This procedure has proved totally reliable in the detection of hemorrhages that are 10 cm or more in diameter Smaller pontine hemorrhages are visualized with less certainty At the same time, coexisting hydrocephalus, tumor, cerebral swelling, and displacement of the intracranial contents are readily appreciated MRI is particularly useful for demonstrating brainstem hemorrhages and residual hemorrhages, which remain visible long after they can no longer be seen by the CT scan (after 4 to 5 weeks) Hemosiderin and iron pigment have their own characteristic appearances, as described earlier In general, lumbar puncture is ill advised, for it may precipitate or aggravate an impending shift of central structures and herniation The white cell count in the peripheral blood may rise tran-.

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Using the Barcode Font in Microsoft Excel (Spreadsheet)
Tutorial in using the Barcode Fonts in Microsoft Excel 2007, 2010, 2013 or 2016. All the functions ... It is extremely easy to create and print barcodes in Excel.

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Inserting a Single Barcode into Microsoft Excel . Switch to the Add-Ins tab. Open the TBarCode Panel . Position the mouse cursor in a cell. Select the barcode type (e.g. Code 128). Enter the barcode data or use the default data for the selected barcode . Adjust the size of the barcode (width, height, module width etc).

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The frequency of a VFO can be adjusted via a varactor diode in the tuned LC circuit Recall that a varactor, also called a varicap, is a semiconductor diode that works as a variable capacitor when it is reverse-biased The capacitance depends on the reverse-bias voltage The greater this voltage, the lower the value of the capacitance The Hartley and Clapp oscillator circuits lend themselves well to varactor-diode frequency control The varactor is placed in series or parallel with the tuning capacitor, and is isolated for dc by blocking capacitors The schematic diagram of Fig 25-7 shows an example of how a varactor can be connected in a tuned circuit The resulting oscillator is called a voltage-controlled oscillator (VCO)

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siently to 15,000/mm3, a higher gure than in thrombosis The sedimentation rate is mildly elevated in some patients Course and Prognosis The immediate prognosis for large and medium-sized cerebral clots is grave; some 30 to 35 percent of patients die in 1 to 30 days Either the hemorrhage extends into the ventricular system or intracranial pressure is elevated to levels that preclude normal perfusion of the brain Sometimes the hemorrhage itself seeps into vital centers such as the hypothalamus or midbrain A formula that predicts outcome of hemorrhage based on clot size has been devised by Broderick and coworkers; it is mainly applicable to putaminal and thalamic hemorrhages They found a close correlation with outcome A volume of 30 mL or less, calculated from the CT scan, predicted a generally favorable outcome; only 1 of their 71 patients with clots larger than 30 mL had regained independent function by 1 month By contrast, in patients with clots of 60 mL or larger and an initial Glasgow Coma Scale score of 8 or less, the mortality was 90 percent (this scale is detailed on page 754) As remarked earlier, it is the location of the hematoma, not simply its size, that determines the clinical effects A clot 60 mL in volume is almost uniformly fatal if situated in the basal ganglia but may be more benign if located in the frontal or occipital lobe From the studies of Diringer and colleagues, it appears that hydrocephalus is also an important predictor of poor outcome, and this accords with our experience In patients who survive ie, in those with smaller hemorrhages there can be a surprising degree of restoration of function, since, in contrast to infarction, the hemorrhage has to some extent pushed brain tissue aside rather than destroyed it Function may return very slowly, however, because extravasated blood takes time to be removed from the tissues Also, since rebleeding from the same site is unlikely, the patient may live for many years In some instances of medium-sized cerebral and cerebellar hemorrhages, papilledema appears after several days of increased intracranial pressure This does not mean that the hemorrhage is increasing in size or swelling only that papilledema is slow to develop Healed scars impinging on the cortex are liable to be epileptogenic; the frequency of seizures after each type of hemorrhage has not been established, but it is lower than for ischemic strokes There is probably no need to administer anticonvulsive medication unless a seizure has occurred The poor prognosis of all but the smallest pontine hemorrhages has already been mentioned Cerebellar hemorrhages present special problems that are discussed below Treatment The management of patients with large intracerebral hemorrhages and coma includes the maintenance of adequate ventilation, use of controlled hyperventilation to a PCO2 of 25 to 30 mmHg, monitoring of intracranial pressure in some cases and its control by the use of tissue-dehydrating agents such as mannitol (osmolality kept at 295 to 305 mosmol/L and Na at 145 to 150 meq), and limiting intravenous infusions to normal saline Qureshi s group has offered data suggesting that aggressive measures to reduce intracranial pressure may be lifesaving and result in good outcome even in patients who have signs of transtentorial herniation In our experience, this type of recovery is exceptional, but treatment may be justi ed in some patients whose medical condition allows it As mentioned, virtually all patients with intracerebral hemorrhage are hypertensive immediately after the stroke because of a generalized sympathoadrenal response The natural trend is for the.

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